part II: SARS-CoV-2 is the Most Dangerous Virus to Ever Exist
Why downplaying the severity of the COVID-19 Pandemic only happens due to Hollywood’s sensationalized depiction of viral outbreaks, and the hidden history of live-attenuated vaccine trials gone-wrong.
Return to part I here.
Albert Sabin was only able to create his temporarily miraculous live-attenuated vaccine against Polio because he noticed that not only could Polio infect the central nervous-system and create the debilitating paralysis that Polio was most known and feared for, but after autopsying hundreds of human cadavers around the Cincinnati area, he found that Polio first infected intestinal tissue before moving on to the nervous system.
And if you were following closely at the start of this pandemic, you might remember that much was made of the fact that SARS-CoV-2 could bind to the receptor ACE2, since it’s expressed a bit in the lungs and would so might facilitate airborne transmission, which somehow was up for debate in the first several years of this pandemic. It was the original SARS-CoV’s ability to bind to ACE2 which scientists assumed gave it the ability to transmit through the air, so in the early months of the pandemic much was made of the fact that SARS-CoV-2 could also bind to this receptor.
However, sometimes only a picture can do reality justice. And if you take a moment to examine the data, you’ll learn that its exceedingly unlikely that ACE2 is the sole receptor that opens the door to airborne transmission, since its in no way unique to our lungs and in fact is far more concentrated throughout our gastrointestinal tracts:
Although it has probably the most lyrical name in our guts, the duodenum isn’t widely known as part of the human gastrointestinal tract, so given the chart above that’s probably worth pointing that out. It’s the third label on the Y-axis, after Small Intestine and Colon, and about a dozen organs before Lungs are listed.
So despite all the celebrity around SARS-CoV-2’s affinity to ACE2 having something to do with its airborne transmission and the many thousands of academic papers documenting how much our friendly novel neighborhood coronavirus just absolutely adores ACE2, it turns out that this affinity is far more likely to be related to wanting to bind with our gastrointestinal tissues - the very same viral nature that Albert Sabin identified to design his almost infallible LAV against Polio.
And in terms of bat-like coronaviruses that bind to ACE2, the first of those was found by a Chinese researcher with the Wuhan Institute of Virology named Xingyi Ge, the lead author of a paper published back in 2013 that would list the WIV’s batwoman, Zhengli Shi, as the final and corresponding author. Then within two years, both of those bat-wrangling WIV all-stars would be co-authors on what would become one of the most notorious research-papers in history, the 2015 masterpiece from Baric’s lab which featured the creation of chimeric coronaviruses whose spike-proteins had been swapped around.
An experiment that you might be able to attach to the development of biological weapons if you’re sporting an extra chromosome of your own and squint really hard, but which makes far more sense as a step in eventually developing a live-attenuated vaccine against SARS-like viruses. After all, Baric’s lab at UNC wasn’t the only known place on earth to be developing SARS-like bioweapons, it was in fact the only place on earth known to be trying to create a live-attenuated vaccine for SARS-like viruses.
And as important as weapons obviously are, humanity’s never actually seen the deployment of a viral WMD or any virus that’s been weaponized at all short of corpses being catapulted over ancient walls - probably in no little part due to the reality that the world is now so interconnected with international travel. It’s not quite literally impossible to imagine deploying an effectively weaponized virus that wouldn’t make its way back to your own population within at least a few years.
But the development of a live-attenuated vaccine against all SARS-like viruses, curing the Common Cold forever? Totally peaceful endeavor, and beyond that - if you stick the landing, many millions of lives are saved, and you’ve just so happened to create the most lucrative viral strain in the history of the world.
Which would explain Xingyi Ge and Zhengli Shi’s coauthorship on that 2015 paper on chimeric coronaviruses out of Baric’s lab at UNC, since although the original SARS-CoV had bound to ACE2 back in 2003, that distinguished it from other known bat-related coronaviruses known to the scientific community at the time especially since it was supposed to come from civets and not bats.
And if the original SARS-CoV was part of an entirely covert live-attenuated vaccine program that leaked out well before it could be weakened but was meant to immunize against the common cold and every other coronavirus known to man, its affinity for ACE2 would also make sense - after all, in the years following its initial emergence SARS-CoV managed to escape from Chinese labs at least four times. In that scenario, its airborne transmission might be a result of other receptors beyond ACE2 that allow for airborne coronavirus transmission along with the instinct for highly-pathogenic viruses of all kinds - but especially coronaviruses - to explore new hosts via airborne transmission.
So just as 2003’s original SARS-Cov and also MERS-CoV were likely part of covert LAV programs that got out way before they were attenuated down into a live-attenuated vaccines, SARS-CoV-2 is what happens when a LAV reverts out of control - something that happened with LAVs against H1N1 in both 1977 and 2009.
Since despite all of its molecular mysteries, the most telling element of SARS-CoV-2 is that after you remove its notorious furin-cleavage site, or FCS, it acts in a completely alien way compared to an influenza whose FCS has been removed. In the case of influenza, if you remove its FCS you get a flu that’s still going to debilitate its host and make active infections like almost nothing has changed, it’s just going to have a hard time jumping into a new species and probably won’t infect nerves as well. But if you remove the FCS of SARS-CoV-2, turns out you get a vastly weakened virus which in fact works as live-attenuated vaccine against further spread of SARS-CoV-2, and won’t even spread among cohoused hamsters.
But just in case that’s not convincing, here’s a bunch more evidence that SARS-CoV-2 began its life as a live-attenuated vaccine:
SARS-Cov-2’s genes are generally more potent immune suppressors than their homologues from the other coronaviruses, including the temporally paradoxical observation that “SARS-CoV-2 proteins are more potent in their inhibition of innate immune signaling than the corresponding SARS-CoV-1 protein.”
SARS-CoV-2 has a novel gene named CaORF15 which encodes a protein which has never been seen before other than in RaTG13, which is a consensus sequence with no natural providence either, and seems to have the immunosuppressive activity that you would expect for a vaccine.
So perhaps it’s a coincidence, but just prior to the emergence of the SARS-CoV-2 pandemic in the summer of 2019, a scientist intimately involved with gain-of-function research named Ron Fouchier made the rather pointed statement below at the European Congress of Virology, in reference to the H1N1 outbreak of 1977 - a virus that wasn’t demonstrated to be artificial until my dad and I published the Clarification that headlines this article in February 2021:
“That’s what happened in the 70s, people were trying to do live-attenuated vaccines and doing human challenge studies and that might be the way the H1 re-emerged in the 70s. Some people say it was a lab accident. I don’t believe that. I think it was actually human challenge studies and live-attenuated vaccines that reverted that are the likely candidates of the 1970 reemergence of H1. And we need to make sure that doesn’t happen again.”
Then by the early months of 2020, perhaps Fouchier’s fears were just remarkably prescient, or perhaps he knew due to his notorious gain-of-function experiments of 2012 with H5N1 which involved an FCS that seemed to appear out-of-nowhere and allow for airborne transmission between ferrets, and due to the history of OPV’s unexpected reversion, just maybe Dr. Ron Fouchier - who rose to notoriety after his 2012 gain-of-function experiments serially passaging H5N1 through ferrets - had a bit of preemptively guilty conscience.
And if Fouchier was aware of covert experiments run by China’s Peoples Liberation Army (PLA) serially passaging coronaviruses, likely at least lightly mimicking his experiments from over a decade ago, that would also explain his statement in this FOIAed email made in the early months of 2020:
At the time, Fouchier’s statement in point two - that a furin-cleavage site (FCS) can appear in human coronaviruses after serial passage - brought a fair amount of confusion among Tony Fauci and his friends who received that email. Because at the time, there was nothing in the published and publically-available literature about any coronavirus gaining a FCS during passage, that’d only been demonstrated during the notorious gain-of-function experiments of 2012 with H5N1 and ferrets, an avian influenza not a human coronavirus.
But - and you might want to hang on as tight as you can to your pearls here - militaries from China to America do in fact operate all kinds of secret scientific projects involving genetic manipulation, especially when they’re related to bioweapons - either their creation or prevention. In the case of SARS-C0V-2 and any artificial chimera that can bind to human cells, there’s always going to be a little of both. Because in their un-attenuated form highly-pathogenic viruses that can bind to human cells well enough for head-t0-head transmission like the original SARS-CoV and MERS-CoV hold clear potential to be weaponized, but after you attenuate a highly-pathogenic virus down into a LAV - like the Rabies virus going from a nightmare death sentence to a miracle after attenuation - there’s no longer any threat, so long as onward community transmission is limited like with OPV around handwashing.
So maybe that’s why it took fifty years for the fact that 1977’s H1N1 variant was in fact engineered to become part of the known and published scientific corpus. Well, kind of, because that paper’s been almost entirely buried despite its link to SARS-CoV-2’s engineered origins. Perhaps that’s a coincidence, or perhaps that’s the result of a concentrated effort to occlude SARS-CoV-2’s origins in yet another LAV program that went sideways.
Ferrets also have an intimate relationship with our ongoing COVID-19 Pandemic and the development of live-attenuated vaccines for SARS-like viruses, as the search for an intermediate species for SARS-CoV-2 was actually solved all the way back in August 2020, when the first peer-reviewed paper arguing for an engineered origin of this virus went out its way to point out that SARS-CoV-2’s immediate affinity for farmed mink had already answered the question of an intermediate host:
2.3. Ferreting Out the Signs of Serial Passage
Curiously, studies examining SARS‐CoV‐2's infectivity in ferrets found that it spreads readily among them, and also appears airborne in that animal model.[ 38 ] This lends support to the idea that ferrets may have been used for serial passage since viruses typically take a significant many months if not years to acclimate enough to spread at all among any new species, nonetheless become airborne, which requires further mutations.
This relationship was further supported by reports out of the Netherlands that the novel coronavirus had spread among thirteen different mink farms there, and also to at least one farm in Denmark[ 39 ] and to another in Spain where 87% of the mink were infected.[ 40 ] Minks are a closely related subspecies of ferret that can produce fertile offspring together, and so the fact that not only did the virus spread to fifteen different farms in three countries, but also appears to have spread from minks into farm workers[ 41 ] indicates that accidental commercial serial passage through minks could have played a role in its creation, as an alternative to laboratory ferrets. Nevertheless, regardless of where any possible serial passage occurred, the fact that SARS‐CoV‐2 spreads from humans to minks and then back to humans demonstrates a high affinity for both species, despite neither nominally being a natural reservoir.
However it’s a solution that’s been completely and entirely buried by the media, and everyone pretending they’re investigating an engineered origin of SARS-CoV-2, which is probably because ferrets are the animal of choice to develop vaccines for SARS-like viruses:
“Although different animal models are used in vaccine studies, the most appropriate model for studying SARS is ferret since it develops the typical clinical signs, viral replication patterns and lung pathology compatible with that of SARS pathogenesis in humans.”
Since the appearance of an FCS in the 2012 gain-of-function experiments came as a surprise to the currently accepted model of mostly linear virology that assumes one set genome moves in near complete stability from host-to-host, it doesn’t come as a surprise at all if you account for virology’s long-lost field: Quasispecies Swarm Theory. A field which was originally proposed by Francis Crick of DNA fame to Dr. Manfred Eigen over an otherwise uneventful breakfast in 1971, and seemed promising given the results already obtained by Sol Spiegelman and colleagues from their serial transfer experiments of RNA taken from a virus that infects bacteria in a closed system, which demonstrated Darwinian behavior in vitro.
A more complicated way of saying that since as far back as DNA was being proposed and accepted, there was a model of RNA behavior which both held up to experimental conditions and also presented a viable framework to help explain how life first emerged from a fractal model of baby-proteins that only build in complexity ever-so-slightly from one stage to the next.
The quasispecies swarm model approaches RNA viruses not as discrete genotypes transmitted on by discrete strains, but instead as quasispecies of mutant swarms of virions which carry distinct but complimentary sets of alleles - collections of genes thought to work together - which cooperate in concert in real-time to establish and expand infections. A concept that can help explain why it’s only farmworkers or others in close and continual contact with live birds who contract H5N1 through the air, a process that that takes multiples waves of infections as the quasispecies swarm looks for the best combination of variants to infect the new host. And so one of the first empirical changes that comes once you consider an RNA virus as a quasispecies genomically, is that at any point in time an average of all the swarm’s variant genomes serves as the smallest selective unit, as opposed to using individual virions or any single extant genome in a population, the classical approach.
When the classical approach is applied to more advanced organisms it seems self-evident, of course each of us has our own discrete genome and pattern of nucleotides that’s unique to who we are as individuals. Right?
Our free will is written into the unique pattern of the nucleotides that makes us who we are.
However infections from RNA viruses don’t work the same way as large organisms being programmed by DNA, and there’s no such thing as an RNA virus that exists out on its own without a whole bunch of its relatives inhabitating neighboring cells. All RNA viruses exists as quasispecies swarms of related variants, sharing related and complimentary genomes just as you share extremely similar genomes with all of your aunts, uncles, and cousins.
So although it’s possible to pluck each of us out away from our families and examine us as discrete units at any one moment in time - Is it really possible to separate any given individual completely and entirely from their family, when you consider the course of their entire lives? So while our lives take many decades to fully take form, RNA viruses go through many generations within just a few hours.
Genetically, at any given moment, sure - your genes aren’t exactly the same as your parents or siblings, assuming you don’t have an identical twin. However just as RNA viruses exist in obvious and observable experiments as collections of related but slightly-different alleles all trying to work together in real time to conquer a new host over the course of hours and days, so do extended families work together to help their close relatives and offer support to accomplish goals - but with humans the timescale is more often years and decades, a reflection of the fact that our ability for reproduction is a nine-month gauntlet to make one copy, as opposed to the viral ability to spit off hundreds of offspring in an hour.
The fundamentally amorphous nature of RNA virus genomes, especially when considered over time, means that the quasispecies approach invalidates the idea of a singular “wild type” isolate genome with one immutable nucleotide sequence described as the contagion at any one moment, since under this approach every RNA virus is by definition a swarming ever-changing mutant cloud of quasispecies variant virions. This results in a probabilistic uncertainty around exactly which section of the cloud is being observed at any one moment, a cloud which will be different the next time you sample it regardless of how representative your first sample was.
As one study revealed, although it’s usually possible to identify a majority consensus sequence from a sample of a host infected by COVID-19, the sample had a broad median variant count of 23, with nearly 250 different variants found in total just within one single host. And considering that about half of the observed mutations thought to have a significant impact on gene expression and samples differing throughout the day even in the same organ system, as well as the fact that barely 2% of the minority variants were found to overlap at all between any two hosts - the inherently nebulous quasispecies swarming nature of SARS-CoV-2 begins to coalesce even more.
And one of the most fundamental premises of quasispecies theory is that unlike the broadly accepted linear theory which imagines that new variants only emerge after recombination or other predictable processes, minority variants can in fact appear seemingly out of nowhere, especially in the context of the swarm recovering from being forced through an evolutionary bottleneck, as occurs with reverting live-attenuating vaccines.
Or in the parlance of quasispecies theory and the inherently cloaked nature of minority variants, its possible for an altered genome to exist within the quasispecies swarm and remain hidden unless you present it with a bottleneck that requires its unique set of skills - such as the airborne jump to a new species which the FCS seems to enable for both influenza and SARS-CoV-2 - at which point it’ll quickly reach fixation.
If that sounds a bit complicated, there’s good news: Back in 2012 the aforementioned Ron Fouchier and another team helped demonstrate this concept with a gain-of-function experiment.
In each of the 2012 gain-of-function serial passage experiments with influenza strains and ferrets, the FCS didn’t appear as a response to the challenge of airborne transmission between hosts, it existed in a very small frequency within each H5N1 swarm prior to each experiment - getting created not by a natural predictable evolutionary process, but instead by the direct artificial outside manipulation of both teams of scientists - and then quickly reached majority status once the bottleneck of jumping from ferret-to-ferret in the air was presented as an evolutionary opportunity.
It was observed by each team after successful airborne transmission between ferrets, however before this challenge was presented to the H5N1 swarms, they were both first heavily mutated by artificial outside means - directly splicing in genes from H1N1 in the case of Dr. Yoshiro Kawaoka, and bathing the swarm in a mutagen in the case of Dr. Ron Fouchier - both artificial, inherently sloppy, and unpredictable processes a long way from surgically splicing precise nucleotides in-and-out. It was these mutational processes which led to the cryptic emergence of the FCS in small minority subpopulations of their swarms prior to their presentation to caged ferrets for passaging.
These were the events that created the FCS during those experiments, the outside intervention of scientists intent on carrying out their gain-of-function experiments - not the challenge of jumping through the air between ferrets. Once it exists anywhere in the swarm, the FCS is going to remain at levels that are too small for typical detection until its special ability is called for: Airborne transmission between mammalian hosts. Directly supporting this specialized airborne role for the FCS is the apparent reemergence of SARS-CoV-2’s FCS within Calu-3 cells - cells grown from the surface of human lungs - after it falls off in Vero cells. The swarm doesn’t need an FCS to flourish inside monkey kidney cells, inside Vero cells - however once it gets placed into human lung cells, now the chance of airborne transmission is back on the table, and so the FCS quickly returns to dominance inside the swarm, reaching complete fixation in just a single passage.
So in 2012 with H5N1, it was caged ferrets who first taught humanity that it just takes one FCS created by unpredictable mutational processes to enable airborne viral transmission. And in 2019 at the Wuhan Military Games it was the 250,000 volunteers from all over China and many thousands of athletes from all over the world crowding into Wuhan, combined with the cultural practices of both pooping in holes and not being overly concerned with handwashing who summoned the FCS back into existence. Which of course is just a fancy way of saying all it would take is a single FCS left behind in the many thousands of doses of a live-attenuated vaccine doses needed for meaningful large-scale testing to eventually reach complete fixation with the SARS-CoV-2 quasispecies swarm, and send the whole thing airborne.
This is probably a good time to mention that the scientist who first promoted hand-washing to prevent the transmission of pathogens lived roughly 200 years ago.
In perhaps the scientific and medical communities’ single biggest warning to anyone who dare rock their boat, Ignaz Semmelweis first proposed back in 1847 that everyone should wash their hands when they went from patient-to-patient, and especially doctors between going from dissecting a cadaver and then shoving themselves forearms-deep up inside birthing women. And yet because he had no theory to explain what seems like the most common sense of practices now, Semmelweis was ridiculed and rejected by the entirety of the scientific community and forced into professional isolation.
Then about 20 years after his assertion, Semmelweis’s voice had been silenced.
Not because it’d been disproven, but because in 1865 due to the strain of arguing against the entirety of civilization and probably seeing lots and lots of corpses every hour of every day for years at a time no matter where he looked and even after he closed his eyes, Semmelweis was committed to an insane asylum. And then within two weeks, he was dead - not technically from the beating he got from the guards, but almost certainly from the gangrene infection that resulted from their beating.
And although live-attenuated vaccines wouldn’t start to revert for a very long time, Semmelweis had also described the single best preventative measure to stop the spread of many pathogens, including the reversion of LAVs. Since although OPV spent decades appearing totally safe, within communities where hand-washing isn’t really a thing or where there isn’t modern plumbing to begin with for any number of reasons, feces will always remain as a specter since they are the most potent viral vector.
Luckily for humanity, Semmelweis’s assertions around the importance of handwashing would begin to start becoming accepted within about 20 years of his death. And in the years that followed he would eventually become known as the “Father of Infection Control” and even the “Savior of Mothers” due to how obviously and immediately deadly unwashed hands could prove to birthing or expectant birthing women especially if they’d recently been shoved up inside a cadaver that’d been dead for months.
So the medical and scientific communities didn’t admit they were wrong in the 1850s without an inordinate amount of kicking-and-screaming, as well as what just about amounted to a homicide of the scientist trying to warn them about how dangerous their practices actually were.
Has much at all changed in nearly 200 years?
Just like the gain-of-function experiments of 2012, the FCS is an unexpected addition whose appearance speaks to the lack of complete scientific knowledge and industrial mistakes, not a weapons system. Was the FCS expected to appear in 2012 with H5N1 and serial passage through ferrets among two different teams in two different parts of the world?
Nope, total surprise, came out of nowhere. Arguably just as much of a surprise to the scientific community as the idea that you should wash your hands after dissecting a cadaver if you were about to shove them up inside a pregnant woman shortly thereafter.
And just like the first deaths due to viral engineering all the way back in 1935 were an accident and not the result of scientific malicious intent, those souls were still helpless orphans slaughtered in the name of science - scientists often don’t set out to kill people, but when they do the first instinct isn’t to apologize, it’s to deflect. Just like the reversion of a live-attenuated vaccine for H1N1 that went global back in 1977 was an accident that became a fifty-year mystery, and just like almost no one - scientist or not - knows that the H1N1 Pandemic of 2009 can be very directly tied to the origins of HIV, as both outbreaks originated as LAVs gone wrong.
But even if you’re having a hard time digesting the reality of where SARS-CoV-2 came from, that doesn’t change any of the results below. All of which indicate not only that this virus not getting weaker, but that taken all together - there isn’t any meaningful difference between what’s happening now and the scenario of HIV going airborne before it could be detected. Because after all, although Hollywood has been overly dramatic when it comes to its depiction of what Evil would look like if it emerged as a viral pandemic, its already nailed the nature of the Devil when it comes to his presence among us.
When the Devil shows up its not with hooves and horns and it’s not with uncontrollable plumes of infected blood. When the Devil shows up and his influence goes unchecked, the only time Evil really has a chance to spread among everyone alive on earth, is when the infestation of his influence is silent and quiet and doesn’t even seem to be happening.
Since one of the things Hollywood got right, is that the Devil’s greatest trick is convincing us that he isn’t even really there at all.
The worst case scenario of a viral infection isn’t a highly-pathogenic and obviously deadly strain like Ebola or the original SARS-CoV, it’s the story that’s already been partially told by HIV. Bombs that beep and make themselves known are possible to defuse, that’s not the worst kind of bomb. The worst kind of bomb is one without a visible public countdown, which won’t be defused because no one even realizes that it’s counting down at all.
So just how much do HIV and SARS-CoV-2 have in common?
Well, both HIV and SARS-CoV-2 have the ability to infect immature red blood cells, causing a drastic imbalance in their level and throwing oxygen levels out of whack - something not observed in other viruses. Building on this mimicry, they also both directly infect and destroy CD4+ T cells, using a similar mechanism - their mass death is the hallmark of HIV’s first ravages. And they share a number of other eerie evolutionary parallels:
“Both viruses evolve using insertions, deletions, and recombination in addition to base substitution. Both viruses evolve under immune pressure and have circulating variants with mutations in key epitope regions that confer relative resistance to neutralizing antibodies; indels are important for the evolution of antibody resistance in both HIV-1 and SARS-CoV-2. Both viruses have heavily glycosylated receptor-binding surface proteins that enable entry into host cells”
Even more alarming, SARS-CoV-2 has begun targeting white blood cells, the immune system’s defenders, by disrupting their basic metabolic functions. Our novel coronavirus has also been observed reactivating endogenous retroviruses within white blood cells, something that also occurs as HIV develops into AIDS, attacking helpful T-cells in a similar fashion to HIV, and also invading our white blood cells without even needing to use ACE2.
And if HIV was linked to CHAT-OPV, that hasn’t been accepted and was barely humored by the scientific community after about 50 years despite plenty of debate. And back in early 2020, no one had even proven that 1977’s H1N1 was engineered at all, nonetheless linked to a live-attenuated vaccine program that had reverted out-of-control. Plus it’s not like anyone was publicly trying to link the H1N1 Pandemic of 2009 to a reverting LAV either, and since reverting LAVs weren’t something the academic world were ever encouraged to investigate or even learn about - The time was ripe for another cover-up.
Starting with the a lead scientific editor with two Pulitzers at The New York Times, so long as the entirety of the media ecosystem was in their pocket - we’d have contact with The Bulletin of Atomic Scientists, CNN, The Washington Post, Pro Publica, and Vanity Fair, however all of them would refuse to admit our work existed at all - all they had to do was wait for SARS-CoV-2 to fade away just like H1N1 in LAV-form had done twice in recent memory. Of course it would get weaker over time just like H1N1, the fabled “Spanish Flu,” had. However even after being engineered by serial passage, H1N1 was still a natural virus carrying natural genes, ones which had been infecting humans for the many thousands of years that influenza has been infecting humanity.
And it’s not just giant organizations like the New York Times refusing to report on the peer-reviewed research pointing at “serial passage” as a primary method used to engineer SARS-CoV-2, even a seemingly well-meaning organization like USRTK will refer to “serial passage” multiple times in its reporting - but not once like to the peer-reviewed papers that explain it, nor even mention that my father and I exist at all. This is not journalism, it's an intentional distraction from any mention of “live-attenuated vaccines” or their past reversions. Even after USRTK’s own FOIA revealed that the head of the DOE’s secretive Z-Division had been discussing our January 2020 whitepaper, I only found that file after stumbling across it on their site - if they were behaving ethically and honestly, why not reach out to us?
Well, perhaps this has something to do with it:
And just in case that seems a bit dated, here’s Claire Danes explaining how the relationship between the CIA and supposedly independent journalists is facilitated right now.
SARS-CoV-2’s viral parallel is OPV, a LAV made from three mutant strains of Polio isolated from samples that’d been extensively passaged through non-human species, and which weren’t ever supposed to be able to cause active infections in humans at all. And although influenzas can have hidden long-term effects that take decades to reveal themselves, no influenza presents with anything like Polio’s paralytic curse. And just like OPV gets stronger the more transmission there is within a community, and just like reverted OPV can blow through the vaccines meant to stop not only natural Polio but even those tailored to stop reverting LAVs - SARS-CoV-2 is quite clearly growing stronger and laughing at vaccines as it blows by them.
Our not-so-friendly neighborhood coronavirus is in no way getting weaker.
On a fundamental molecular level, SARS-CoV-2 has been getting more thermodynamically stable as it reverts back closer-and-closer to its original strongest form, allowing initial infections to more easily penetrate deeper into host bodies. And this thermodynamic stability also translates into structural stability, allowing the virus to exist for longer and longer on surfaces, as the newer Omicron variant lasts between three and five times longer than earlier strains depending on the surface they’re being tested on.
This is correlated with how much stronger our novel coronavirus is compared to the flu, since while influenza is inactivated just floating around in the air after just thirty minutes it takes SARS-CoV-2 days to become inactivated in typical commercial air. A phenomenon that likely contributes to the fact that the Omicron variant is now seven-times more lethal than influenza in Hong Kong’s children.
And while the virus is growing sturdier, it’s also regaining more of its highly-pathogenic capabilities and gaining the ability to infect new mammalian hosts. Newer variants are beginning to show the ability to infect hosts ranging from mice, marmosets, and koalas all the way to rats and civets. Jumping into rats is an inevitability that will turn every urban sewer on earth into white-hot forges for new swarming variants, other mutations waiting to happen have the potential to increase binding by one-thousand fold. And beginning to explore a jump into civets is a reminder that they were incorrectly identified as the original host for the first SARS - all samples jumped from humans into civets, not the other way around - a chimeric virus with roughly a 15% mortality rate.
There is nothing slowing this reversion down, the futility of all the vaccination programs is even more apparent than it was a year ago, as new studies “suggest that SARS-CoV-2 is diverging into distinct serological phenotypes and that vaccines tailored to one variant may become vulnerable to infections with another." And due to the emergence of new serotypes, the first step for ADE, it already appears as if troublesome infection-enhancing antibodies are emerging in spike-protein gene-therapy recipients who are exposed to newer variants - while ADE didn’t appear with exposure to the original Wuhan strain, even just the Delta variant was already beginning to use ADE against us.
This means newer, stronger variants will inevitably produce more ADE within vaccinated populations, all the while the Biden Administration very intentionally chose political expediency instead of public health, embodying the Nazi ideal of telling its victims they’re just headed off to the showers as part of their normal daily productive routine - when really the system they’re a part of is dictating their eventual and inevitable execution.
There’s a very good reason that the worst crime anyone in the media is willing to even vaguely humor at all when it comes to the origins of SARS-CoV-2 is the idea that maybe NIH had somehow managed to accidentally fund the gain-of-function research that eventually led to a virus that may have leaked out - totally by accident and with no intention by anyone at all, and certainly not with any intent from China. The finger is pointed at Tony Fauci as if he’ll be able to hold the weight of hundreds of millions of innocent souls lost too early, but then if he gets a pardon he’s untouchable and on paper the cover-up is airtight.
Airtight, at least so long as there’s no one busy poking holes in the entire thing.
So despite the fact that the State Department’s Arms Control team was looking to use our August 2020 peer-reviewed paper on serial passage as early as November 2020 to try and invoke the Biological Weapons Convention against China, we didn’t hear from anyone on that team - or from anyone officially linked to our own government at all - until a couple weeks into April 2021, after I’d published this site’s pinned article on April 1st, where I first argued explicitly that this pandemic began when an orally-administered live-attenuated vaccine against SARS-like viruses first conceived at Baric’s lab in UNC and created as a joint PLA-DARPA project began to revert out-of-control at the Wuhan Military Games.
Because there are plenty of scenarios a lot worse than the idea of a virus that was carelessly funded and which then leaked out somehow, but definitely completely and entirely by accident. Because if SARS-CoV-2 did begin as a live-attenuated vaccine designed and tested in secret by the PLA, well then of course it was polished on China’s Uighur concentration camps - now that orphans and invalids and prisoners and Africans are off-limits for immoral experimental scientific testing for the multi-billionare dollar pharmaceutical industry, do you really believe that the Uighurs would be left unvictimized?
By the biggest authoritarian government on the planet, as a captive and utterly helpless population?
And since Uighurs are Muslims who always wash their hands after defecation, and bathe many times a day by default, what might’ve seemed like the successful test of a live-attenuated vaccine among the Uighur concentration camps would’ve turned into humanity’s last gong-show at the Wuhan Military Games where 250,000 Chinese volunteers would’ve been pooping in open holes and washing their hands much less than half the time. So a virus that seemed totally safe in one scenario, among Muslims who wash fastidiously, would’ve quickly begun to transmit and revert within an overcrowded urban scenario among a population that’s used to pooping in an open hole and only sometimes washing their hands afterwards.
This was in fact the very first scenario that resulted in engineered viruses killing people at all, untested poorly designed LAVs being tested on innocent populations, in experiments that have remained almost entirely undiscussed within the scientific literature for nearly a century. And if anyone with any sort of virological background at all was being even the tiniest bit honest about a discussion around engineered viruses that could infect humans all across the globe, they would be starting with reverting live-attenuated vaccines.
After all, right now at this very moment there’s only one kind of virus that’s definitively known to be engineered that’s infecting humans all over the earth, and that’s the same sort of engineered virus that’s already been linked in the scientific literature to global pandemics twice in just the past fifty years. But despite the best efforts of a whole bunch of folks, just about all of whom have existing ties - either direct or financial - with the U.S. Government, and all of whom have refused to publicly admit that our peer-reviewed work on “serial passage” exists at all, the discussion around reverting live-attenuated vaccines hasn’t remained entirely buried.
Confronting the idea that SARS-CoV-2 began as a live-attenuated vaccine would require the scientific and academic communities to betray the very natures that they’ve been exhibiting for nearly a century, when experimentation on helpless victims was written-off as expediency - putting the useless to at least some kind of use. Behavior that would continue throughout the AIDS Pandemic, when none other than Tony Fauci was rolled-out to both demonize the afflicted and present a cure that didn’t work at all.
To test the first live-attenuated vaccines, human subjects had to be found, and so American scientists flocked first to orphanages to find victims whose death and disappearance no one would notice. But then when our laws got updated enough to make experimenting on orphans and prisoners and anyone else who’d been institutionalized and forgotten illegal, such as the Willowbrook State School whose heinous experiments on vulnerable kids were revealed in the early 1970s by Geraldo’s expose, that pool of subjects dried up. Then finally with the exposure of the heinous Tuskegee Syphilis Study and MK-ULTRA, it seemed like immoral government-sponsored human experimentation might finally be over.
But then unluckily for all of us, men like Bill Gates didn’t let Tuskegee stop them, and moved on to using Africa as one giant pharmaceutical testing ground in the decades that followed, and so experimental drugs and vaccines were pumped into the continent without anything even vaguely resembling ethical safety controls. And when a company couldn’t afford to get its testing done all the way over in Africa, then there were always plenty of African-Americans and other disenfranchised minorities right at home - slipping through the cracks and just waiting to be double-blinded!
There are few people alive more interested in vaccine development than Bill Gates, perhaps because after feeling like he’d pretty much conquered the digital world of ones and zeroes by the late 1990s, there was nothing left for him to do but apply his genius to the genetic world of nucleotides: As, Ts, Gs, and Cs.
Not binary like the digital world, but perhaps close enough.
So perhaps its just one big unfortunate coincidence that it's been Gates’ Global Polio Eradication Initiative, heavily funded by Gates since the 1980s, which first called attention to the need for an improved version of OPV and which created the nOPV which… didn’t work either, and still reverts all the way back to virulent Polio. Because there is no one alive better positioned to liaison between the Chinese government and secretive elements within the American government like DARPA, which perhaps is why Gates was present at Event 201 which took place in NYC in the fall at 2019 right as the Wuhan Military Games were occurring, a date multiple lines of evidence point to as the actual start of the COVID-19 Pandemic.
And Event 201 was also attended by Avril Haines, who was then Deputy Director of the CIA, and would soon become head of the entire Intelligence Community as the Director of National Intelligence. Which is the position she held in August 2020, when our peer-reviewed paper was published, and then within a couple weeks Haines had long-time DARPA and Intelligence Community consultant Richard Danzig email me and my dad to try and get that paper retracted. An email whose distrubtion included luminaries such as Johns Hopkins’ Gigi Kwik-Gronvall - one of the loudest proponents against a lab origin.
It’s probably a good time to point out that Johns Hopkins and DARPA are many hundreds of millions of dollars deep in each others’ business.
When Richard Danzig emailed us we had no idea who he was, but a quick googling revealed that in 2018 he was still very much involved with DARPA and the Intelligence Community, and just a couple years before the pandemic was coauthoring papers alongside Avril Haines pitching the idea that a pandemic would mean a truce between China and America:
This global environment requires that a successful American security strategy do more than attempt to prevail in or manage competition; it must artfully blend and balance competition and cooperation. This is particularly evident in our relations with China, a nation whose economic power, we have emphasized, is approaching America’s and will probably come to exceed it. In this context, our recommended strategic premises encourage a willingness to take risks for peace that at least approaches our acceptance of risk in conflict. This example provides some illustrations of how our call for cooperative initiatives might be translated into action.
…
So it may be in the years ahead. If, for example, a highly contagious disease like SARS broke out in North Korea, we believe the resulting concerns would render America and China, at least for these purposes, allies.
So given that co-authorship between Danzig and Haines, and given Danzig’s uninvited contact with us, its worth taking a moment to take a look at some of the names Danzig acknowledged in his May 2008 paper Preparing for Catastrophic Bioterrorism where he’s specifically listed as a consultant to DARPA and “other Federal Agencies.” Among the handful of names Danzig lists in that 2008 paper is Bob Kadlec, who at the time was just a member of the White House Homeland Security Council. However by 2023, Kadlec had advanced quite a bit, since he was the Assistant Secretary for the US Department of Health during the pandemic.
So if Richard Danzig had a pre-existing relationship with Bob Kadlec dating back to 2008, and if Danzig had co-authored a geopolitical security paper alongside Avril Haines as recently as 2018 - Is it really a huge stretch to imagine that both Avril Haines and Bob Kadlec have been following along with all of this for the past few years?
What are the chances that Richard Danzig contacted us - the authors of the first peer-reviewed paper arguing for a lab origin of SARS-CoV-2 which pointed directly at the serial passage that defines live-attenuated vaccines - and that Haines and Kadlec and everyone else he’s been working with inside DARPA for decades were completely and entirely in the dark about all of it?
Those odds are pretty much zero, especially when you consider how Kadlec describes the genesis of this pandemic:
Some within the United States Government – including Kadlec – now suspect it was vaccine research by People’s Liberation Army scientist Zhou Yusen that may have led to the pandemic. Like Ben Hu, Zhou was also close to Shi Zhengli. Zhou was a decorated Chinese military scientist who worked on coronaviruses at the Beijing Institute of Microbiology and Epidemiology, which is part of the Chinese Academy of Military Medical Sciences and sits under the control of the People’s Liberation Army. His work for the Chinese military was extensive and he often worked alongside the Wuhan Institute of Virology and Shi Zhengli.
…
Perhaps no one has analysed more of Zhou Yusen’s papers than Bob Kadlec, one of the top health officials in the US administration at the time of the pandemic. “We think vaccine research resulted in the pandemic – that vaccine research was the proximate cause,” he tells this magazine.
Kadlec notes that the type of coronavirus vaccine research Zhou Yusen was involved in was “very high risk” because it involved infecting live animals, administering some of them with the vaccine and using others as a control, and then keeping them alive for a period of days or even weeks before eventually euthanising them.
And so, if events were spinning out of control in the aftermath of the Wuhan Military Games in the fall of 2019, where 250,000 extra volunteers gathered in addition to all the international athletes and the poor sanitation of temporary porta-potties at best and pooping in holes by default super-charged the reversion of SARS-CoV-2 just like it does with OPV. So if just one single FCS had been left behind during the industrial attenuation and production process of a LAV meant for thousands - if you take quasispecies theory and the ability of even the smallest minority variant to reach fixation into consideration, you’ll understand that’s all it would’ve taken to send SARS-CoV-2 airborne and begin the COVID-19 Pandemic.
So if something like that was going on, how do you think Bill Gates would’ve been spending his time and energy, and whole epic fuckloads of his money:
What followed [the start of the pandemic] was a steady, almost inexorable shift in power from the overwhelmed governments to a group of non-governmental organizations, according to a seven-month investigation by POLITICO journalists based in the U.S. and Europe and the German newspaper WELT. Armed with expertise, bolstered by contacts at the highest levels of Western nations and empowered by well-grooved relationships with drug makers, the four organizations took on roles often played by governments — but without the accountability of governments.
The four organizations had worked together in the past, and three of them shared a common history. The largest and most powerful was the Bill & Melinda Gates Foundation, one of the largest philanthropies in the world. Then there was Gavi, the global vaccine organization that Gates helped to found to inoculate people in low-income nations, and the Wellcome Trust, a British research foundation with a multibillion dollar endowment that had worked with the Gates Foundation in previous years. Finally, there was the Coalition for Epidemic Preparedness Innovations, or CEPI, the international vaccine research and development group that Gates and Wellcome both helped to create in 2017.
While dozens of global health organizations participated in the world’s response to Covid, the POLITICO and WELT investigation focused on these four organizations because of their connections to one another — both Gavi and CEPI received seed funding from the Bill & Melinda Gates Foundation — and because they together played a critical role in advising governments and the WHO.
The WHO was crucial to the groups’ rise to power. All had longstanding ties to the global health body. The boards of both CEPI and Gavi have a specially designated WHO representative. There is also a revolving door between employment in the groups and work for the WHO: Former WHO employees now work at the Gates Foundation and CEPI; some, such as Chris Wolff, the deputy director of country partnerships at the Gates Foundation, occupy important positions.
Much of the groups’ clout with the WHO stems simply from money. Since the start of the pandemic in 2020, the Gates Foundation, Gavi, and the Wellcome Trust have donated collectively more than $1.4 billion to the WHO — a significantly greater amount than most other official member states, including the United States and the European Commission, according to data provided by the WHO.
“You have to remember that when you’re dealing with the Gates Foundation, it’s almost like you’re dealing with another major country in terms of their donations to these global health organizations,” a former senior U.S. health official said.
So all that said, the bad news for humanity is that there’s quite literally no one with significant power and a voice interested in doing anything more than at most distracting from the truth.
The only reason anyone would want you to focus on bat-caves at this point is to distract you from the fact that although SARS-CoV-2’s distant ancestor’s hosts were bats, when it first hit the world it only spread among two species: humans and the farmed mink which are overlapping species with the lab ferrets used to attenuate LAVs - it barely bound to bat cells at all and never spread from people to them. Simply asserting that SARS-CoV-2 was engineered and leaked out somehow by accident, without first accounting for the long and ongoing history of reverting live-attenuated vaccines infecting humans all across the globe for almost a century, only gives the Devil more and more material for his disguise.
After all, Satan’s greatest trick is to distract you from the reality that he could even be there at all. Sure bad things happen, but those are mostly accidents - not the result of intentional human malice.
And it’s no coincidence at all that there’s an enormous number of folks insisting that this virus is both engineered and that it’s not really a threat, and that infection shouldn’t be seen as any sort of big deal. Turns out that if they aren’t virologists who have spent the past several years acting like there’s no way “serial passage” played a role in its creation, then they often have ties directly either with the billionaires whose existence depends on keeping the economy chugging along like nothing is wrong, or with DARPA if you bother to google their names - from David Relman to Alex Washburne - the entire discourse around direct engineering and accidental leaks is a very intentional distraction.
A distraction that got its start all the way back in early 2020 in an almost simultaneous genesis with this pandemic, when on a brisk Thursday afternoon, the former head of Harvard’s Chemistry Department, Dr. Charles Lieber, strode out of a Boston federal courthouse after agreeing to pay a $1 million cash bond, surrender his and his wife’s passports, and disclose any foreign bank accounts.Lieber had been arrested by the FBI just two days early on January 28th, 2020, on charges that he’d made false statements to Department of Defense and Harvard investigators looking into his ongoing ties with the CCP’s Ten Thousand Talents program, including collaboration with ongoing projects in Wuhan and collecting millions of dollars from the Chinese government for his assistance.
In the confusion around the pandemic’s emergence, the arrest of a Harvard chemistry professor with loose ties to the city of Wuhan certainly seemed strange, but it didn’t seem to have any direct involvement. When samples of SARS-CoV-2 were first being released to the world they seemed remarkably similar, and given how much they spread over the wet market, certainly didn’t seem to be hiding or hard to detect.
So maybe a professor with profound ties to China who’d pioneered research into using electromagnetic fields and nanotechnology to simultaneously detect individual viruses getting arrested at first appeared likely to be coincidental. At the time of its creation in 2004, Lieber marketed the nanotechnology as an invaluable tool for the military to detect biological weapons, and in the years that followed it was presumably privatized and patented since this virus-detecting nanotechnology and Dr. Charles Lieber both just about disappear from the internet until his arrest in January 2020.
So there’s no way of knowing exactly how fine-tuned Lieber’s nanotechnology became in the fifteen years before the COVID-19 Pandemic began, since when it was first announced in 2004 it was only mentioned as being able to distinguish between different families of virus – such as influenza or adenovirus, H1N1 or Dengue Fever. However in all that time, it seems plausible that Lieber’s technology was refined to be able to detect the difference even between different classes of influenza strains. And from looking at one of the patents, it sure seems like it might’ve advanced to the point where it would be able to remotely detect individual nucleotide variations.
And its hard to imagine that Charles Lieber’s release would’ve happened without the intervention of one of the co-founders of MIT’s Broad Institute, Stuart Schreiber, who organized a letter meant to rally support for poor downtrodden Dr. Lieber. Whose only crime was secretly accepting millions and millions of dollars from an adversarial foreign government with an extremely questionable past of unethical genetic experimentation, and which is almost openly committing an institutionalized genocide of an ethnic minority.
Apparently one of the co-founders of the Broad Institute wants his good buddy Chuck back on Harvard’s payroll and reinstated, since if the Department of Justice keeps being mean to preeminent American scientists who secretly accept millions of dollars in bribes for forwarding the interests of a foreign government and then lying about it – other American scientists might not collaborate with Chinese scientists. And if other American scientists aren’t accepting bribes from the CCP and if the actual origins of this pandemic are exposed asgain-of-function research, how is the Broad Institute ever going to make sure its brand-new program which just got a $32 million grant from DARPA is able to carry-out the dangerous and unpredictable gain-of-function research on the human genome it has planned - if there’s another moratorium against gain-of-function work even vaguely touching on the human genome?
Although none of its copy mentions manipulating viruses by name, MIT’s Foundry makes it very clear that tinkering with the human genome is a priority – nominally to improve it of course – however with fully 10% of the human genome derived from one virus or another, including a gene that appears to be fundamentally involved with memory which uses a virus-like action, the idea that the full gamut of experiments on the human genome can be run without being able to serially passage viruses to fully illuminate their full range of interactions with our genome is absurd.
So the world’s billionaires are depending on you being a very particular flavor of stupid, to ignore the fact that the Broad Institute’s Alina Chan’s cartoonishly flattering biopic in Boston Magazine was published less than three-weeks after my father and I first got published in a peer-reviewed publication arguing extensively about the fundamental role serial passage must’ve played in the genesis of SARS-CoV-2. In the years since, Alina Chan has acted like she’s some kind of downtrodden victim despite being quoted many times in countless articles, giving dozens of interviews, and writing a book.
All of that despite never passing peer-review and being a markedly junior scientist, especially compared to my father’s career across multiple decades including being GenBank’s longest serving scientist when he retired right before the pandemic. And following that same pattern, my father and I didn’t were only contacted by a government employee about all of our work after I published Golden Silkworms in Pandora’s Box on April 1st 2021, where I explicitly argued this pandemic began when an orally-administered SARS-like LAV designed with the help of Baric’s lab at UNC started to revert out-of-control at the Wuhan Military Games.
This is despite the fact that David Asher had been aware of our peer-reviewed work since at least November 2020, and yet waited all those months - until I explicitly began writing about a reverting LAV - to contact us at all.
And then after Asher failed to silence us, a few months later in the fall of 2021 the “DEFUSE” proposal was “leaked” by a DARPA employee pretending to suddenly get a streak of integrity for some random reason nearly two years into the pandemic, and all of a sudden that became the only possibly explanation for an engineered virus. A proposal that wasn’t even written until August 2021, and that described the creation of a LAV designed to infect bats - not humans. And although SARS-CoV-2’s ancestral genome appears to have come from bats at some point in the past, our friendly novel neighborhood coronavirus hardly infects bat cells at all, and has never infected bats outside of a lab where they can be completely bathed with the virus. In the first months of this pandemic, SARS-CoV-2 only transmitted among two species: humans and the mink who are sister species with the lab ferrets used to attenuate LAVs.
In the meantime, Alina Chan would be busy attempting to bribe other scientists to have them remove me and my dad from another joint paper we’d been working on about the origins of this pandemic.
Why is it that the American government and the entirety of the media is very excited about interviewing and quoting Alina Chan, an employee of an organization that’s already been linked to Jeffery Epstein’s infiltration and corruption of America’s government, but has never once quoted or interviewed me or my dad - despite our two peer-reviewed publications, the second of which resolved a scientific mystery that’s almost fifty years old?
Well for an entire multitude of reasons, the American government has also managed to keep Jeffery Epstein’s client list and obvious relationship to our intelligence agencies pretty much out of public view or discussion. And just in case it seems a little bit out of left field to suddenly invoke Jeffery Epstein’s name and crimes, there are in fact an enormous number of ties linking Jeffery Epstein not just to MIT, but also to the Broad Institute in particular.
For example, while another one of the Broad Institute’s cofounders was fighting for Stuart Schreiber’s adjudication, here’s why Eric Lander - another Broad Institute cofounder and Biden White House Director of the Office of Science and Technology Policy until 2022 - couldn’t get himself confirmed in Biden’s Cabinet, followed by a picture that should help color in some of the edges:
Lander is the director of the Broad Institute of M.I.T. and Harvard and Biden’s pick to be director of the Office of Science and Technology Policy, which the president for the first time has made a Cabinet-level post. He’s the only Cabinet nominee who has yet to be confirmed (Biden has not yet nominated an Office of Management and Budget director after he had to withdraw his first pick). His confirmation hearing is set for April 29, more than two months after the hearings for Biden’s other Cabinet nominees wrapped up.
Lander and several other professors met with Epstein in 2012 in the office of Martin Nowak, a Harvard mathematical biologist, four years after Epstein pleaded guilty to solicitation of prostitution involving an underage girl. The meeting was reported by BuzzFeed News as part of a 2019 investigation that revealed Epstein’s lavish donations to scientists at M.I.T. and Harvard. There are also several photographs of the meeting showing Epstein with scientists, including two of Epstein and Lander. The pictures had previously been on JeffreyEpstein.org, a website Epstein ran for one of his foundations.
And since those telltale photos of Epstein and Lander appear to have been pretty thoroughly scrubbed from the internet, here’s one that gets the same message across:
And although the insistence that SARS-C0V-2 is entirely tame is the worst kind of atrocity, at least it’s more transparently evil than pretending there’s no way to know how bad it is at all.
That’s not to say everyone involved in the obfuscation understands exactly what they’re doing, far from it. That’s the entire idea, most folks without billions of dollars haven’t sold their souls yet - they’re not intentionally helping the perpetuation of Evil, just as none of the scientists involved with the testing on helpless orphans and prisoners over the past many decades thought they were doing Satan’s deeds. Long before Milgram, they proved that well-meaning people would do all sorts of heinous shit in the name of scientific progress.
So just so you’re aware, it’d be real easy for the CIA to do what it’s done over-and-over again for decades, and spread its tentacles around enough to convince a bunch of misguided Americans and greedy foreign-nationals that hiding the truth about the origins of SARS-CoV-2 was in America’s best interest, and that helping to occlude our involvement in any way with a virus that got out, that spinning those lies would somehow would make them patriots.
Effective Evil is a quiet thing that silently twists our hearts with the illusion of well-meaning.
The CIA quite literally makes its living convincing people put in places of trust to do really heinous acts of violence and betrayal. People throughout history who believed they were serving the public good, because of course their ends justified their means.
A relationship between intentions and outcomes that was likely noticed shortly after genetic engineered as a concept entered the public consciousness, since it didn’t take long after the manipulation of nucleotides emerged for parallels to be made to the inherent prohibition against “playing God” that’s long been understood to be symbolized within the first tree that shows up in the Book of Genesis. A tree that most people don’t realize bears two simultaneous and interwoven names: The Tree of Life as well as The Tree of the Knowledge of Good and Evil. So given the power that nucleotides gives those who manipulate them over the very fabric of all life that exists, it shouldn’t come as any sort of a surprise that genetic engineering has so long been linked with just about the oldest moral, ethical, and religious warnings that exists.
Because scientists haven’t gotten everything right, but at least before long they realized that genetic engineering poses a threat not just to a few civilizations gathered around any particular basin or other geographical feature, but a clear and present danger to all humanity - as a very species. Because if Evil is allowed to spread unchecked, if Satan’s mask never slips because there’s just one set of hands tugging at it, if the day does come when the courage of men fails, when we forsake our friends, and break all bonds of fellowship - that’s when Evil wins and Satan prevails.
But there’s some good news, and in addition to illuminating the cloaked nature the Devil seeks when he takes human form, Hollywood definitely nailed what it means to fight back against Evil. So despite the fact that we all wish this pandemic hadn’t come to us, all we have to do is decide what to do with the time that is given us. Things have seemed all wrong, and by rights none of us should even be here. But we are. And maybe in the end, it’s like we’re in the great stories - the ones that really mattered, full of darkness and danger they were. And sometimes you didn’t want to know the end, because how could the end be happy?
How could the world go back to the way it was after so much bad had happened?
The stories that came down to just one person, no matter how small or uninfluential they think they might be, making a choice to do the right thing, and doing whatever they can to fight back against the Evil that’s threatening to consume us all.
Because make no mistake, the clock is ticking and our time may very well be coming to an end.
Our current existence as a species is in fact no different than the first few years of the theoretical scenario of HIV going airborne, and slowly consuming humanity’s collective ability to fight-back against pathogens we easily fend off while healthy. You’ll find an enormous amount of evidence for that below, and perhaps the most you can do is link one more study with more evidence of SARS-CoV-2’s insidious effects, comments are open to everyone so don’t be shy.
But for some of you, maybe that’s not where your story ends.
No way to know until you wake up tomorrow and decide to turn yet another page, but perhaps taking a little more time to notice all that your ancestors have already written on the pages that have come before, and all the warnings that they’ve been attempting to leave you.
The Obfuscation around and Hidden Effects of SARS-CoV-2:
Hospitals are no longer required to report COVID cases caught inside of them in April 2023
EU trying to normalize mortality data to hide it.
SARS-CoV-2 can create silent infections that don’t leave antibodies behind.
Wastewater sampling in Canada suggests COVID case rate 19 times higher than reported.
Long Covid can develop without ever testing positive.
Can cloak itself from white blood-cells using genes also used by HIV.
Kills nearly one-third of patients within a year after they leave the ICU following treatment.
Nearly 80% of infected kids eventually developed a new health condition.
Kills children while misclassified as SIDS.
Appears to be accelerating the growth of several types of cancer all across the world.
Unusual child deaths are spiking.
Outbreaks of antibiotic-resistant bacteria are increasing.
COVID creates long term silent damage in multiple organ systems.
Teenage math and reading skills in an unprecedented decline all across the world.
In June 2023 there was a surge of abnormal brain abscesses in children.
It can persist and grow in a wide range of hidden tissue reservoirs.
Cases of measles are spiking in Japan.
Whooping cough are spiking in England.
Toxic shock syndrome is growing at an unprecedented rate in Japan.
Military pilots reported 17-times more medical incidents during the pandemic.
Decreased performance in one-quarter of infected Marines.
Tuberculosis in America is increasing after a 30-year decline.
Measles is spiking all across America.
Maternal mortality increased 80% from 2020 to 2021 in Pennsylvania.
25% of students in Ohio were chronically absent in 2023.
Infection increases your odds of a car crash as much as drunk-driving or having a seizure disorder.
Tuberculosis in Scotland surged by 40% in 2023, the largest increase ever recorded.
Long Covid is already affecting nearly one-quarter of the population.
SARS-CoV-2 is Getting Stronger
New variants get better-and-better at evading our immune system.
Newer variants are more neuroinvasive than older ones.
There’s been an evolution of enhanced innate immune suppression by SARS-CoV-2 Omicron subvariants
Omicron is far better at infecting noses than earlier variants.
Newer variants have more structural stability and resist degrading more.
Infection by old variants provides almost no protection against new variants.
Newer variants have increased immune evasion and spread, and are more immune resistant.
The newest variants are the most resistant to antibodies and spread the most effectively.
SARS-CoV-2’s Influence on Autoimmunity and Cancer
It reactivates endogenous human retroviruses like Herpes.
Triggers molecular pathways known to lead to cancer formation.
Raises your risk of developing multiple sclerosis
Causes a long-term reduction in all of your innate and adaptive immune cells.
Productively infects all the primary human immune cells.
Predisposes you to staph infections.
Decreased T-cell responses by more than 75% in about 75% of cases.
Binds directly to white blood cells.
Causes immune changes known to lead to Schizophrenia.
Activates the expresses of endogenous retroviruses that cause inflammation and disease.
Increases the risk you’ll develop herpes.
Damages children’s immune systems by impairing defensive cells.
Interacts with multiple pathways known to eventually lead to cancer over the course of years.
Causes immune cells to age more rapidly and can kill protective T-cells.
Long Covid is linked to the death of T4 cells, disarming the immune system.
Triggers a latent virus in the oral mucosa that’s linked to Chronic Fatigue Syndrome.
Caused a surge of a multidrug-resistant fungal pathogen in Israeli hospitals, and in American ones.
Reactivates the Epstein-Barr Virus which leads to Long Covid.
Suppress innate immunity by targeting mitochondrial defenses.
Makes colon cancer cells more invasive.
Infects a range of lymphocytes, the cells also destroyed by HIV.
Persists in T-cells for at least two years.
Increases autoimmune and connective tissue disorders.
Creates long-term damage to the immune system.
Long Covid is linked with T-cell dysregulation and an uncoordinated adaptive immune response.
Can supercharge cervical cancer by removing immune pressure on precancerous cells.
Promotes the malignancy and growth of breast cancer cells.
Increases the rate of pancreatic cancers.
Accelerates the development of Type I diabetes in children predisposed to it.
Over 40% of patients worldwide had a coinfection with drug-resistant organisms.
Rapidly kills off protective white blood-cells.
Increased the rate of autoimmune hepatitis in kids by over three-times.
Antibodies meant to fight against it rapidly disappear from bone-marrow.
Genetic Disruptions from SARS-CoV-2
Dysregulates your immune system by changing the way its genes are expressed.
COVID-19 accelerates biological aging by accelerating your epigenetic clock.
SARS-CoV-2 alters the scaffolding of your DNA.
Changes the gene expression of white blood cells to increase clotting and cause dysfunction.
Interacts with cancer-causing genes.
Increases intestinal permeability and inflammation while attacking your healthy bacteria.
SARS-CoV-2’s Neurological Damage
Alters your brain structure to make you dumber.
Makes brain cells fuse together into clumps that impair brain function.
Mirrors the effects of HIV against the brain.
Infects neurons then migrates throughout the brain.
Causes persistent structural, functional, and cognitive changes in key areas of young-adult brains.
Creates brain-damage that makes it look like your brain has aged 20 years.
Brain damage and cognitive complaints continue even two years after infection.
Damages your brain with the same pattern that high-altitude disease does.
Creates brain damage that isn’t detectable by typical tests.
Attacks and infects multiple types of brain-cells leading to long term damage.
Causes brain-damage in infants infected during pregnancy.
Accelerates the progression of existing brain tumors.
Damages small cerebral blood vessels causing neurological symptoms.
Can create a silent and deadly “happy hypoxia.”
Caused increased memory and concentration problems.
Eats more of your brain the more previous exposure to other coronaviruses you’ve had.
May cause persistent dysfunction in brainstems.
Caused a spike in autoimmune encephalitis.
Causes abnormalities in cerebral spinal fluid that are linked to cognitive dysfunction.
Created a worldwide surge in a vast array of psychiatric disorders.
Leads to neurotoxic clumps of proteins known as amyloids also linked to Alzheimer’s and Parkinsons.
Can lead to Lewy body dementia.
Directly alters your brain architecture.
Creates the same structural neurological destruction as Alzheimer’s.
Create lasting damage to mitochondria, the powerhouse of the cell.
Leads to overlapping nervous system disorders indistinguishable from those caused by HIV.
Directly infects brain marrow and can surf along the central-nervous system.
Accelerates brain-aging in middle-aged adults.
Creates a wide range of neurological and mental health problems in kids.
Changes the shape of your hippocampus and causes anxiety and depression.
Led to abnormal brain scans in over 40% of kids worldwide.
Infections increase the rate of dementia, depression, anxiety, and disturbed sleep.
Gastrointestinal Issues Linked to SARS-CoV-2
Leads to fatal gastrointestinal disorders that kill you via sepsis.
Sheds through feces for at least seven months after infection.
Colonizes your intestines since they express more ACE2 than any other organ.
Newer variants create increased fecal shedding.
Heart and Lung Complications from SARS-CoV-2
Can activate latent Tuberculosis.
Can lead to clotting issues so bad that amputation is needed.
Heart attacks in young adults are up 30% over the expected rate
Creates pulmonary dysfunction in 25% of patients for at least a year.
Results in a high risk of heart tumors.
Can persist in the lungs for at least 18 months after infection.
Leads to a higher risk of heart failure even without previous heart disease.
Persistently infects heart tissue and leads to an increased risk of stroke and other dysfunction.
Leaves 75% of young adult patients with a cardiac injury visible on MRI.
90% of kids with Long Covid have decreased cardiovascular function.
Infections double your risk of suffering an adverse cardiac event, from stoke to death.
Reproductive Problems from SARS-CoV-2
Reduces the quality of semen for months
Creates alterations in the placenta that lead to pregnancy complications.
Drastically decreases semen DNA quality.
Mothers transmit it to their fetus, where it causes organ abnormalities.
Caused neurodevelopmental impairment in about half of infants.
Infected mothers alter the neurodevelopment of their babies.
Accelerates the rate at which erectile dysfunction worsens.
Increases the chances your baby has a malformed brain.
Damages fetal brains in their first trimester, increasing the odds of a brain-bleed.
Infected mothers have babies with a ten-times increased chance of developmental delay.
Impairs male fertility by targeting semen quality and testosterone levels.
Increases localized placental damage.
Babies born with heart defects are up 16% during the pandemic.
Credit to tern for many of the charts above.
Wow...this is an epic amount of information! If I wasn't already, I am officially freaked out now. No idea what can be done at this point since the genie is out of the bottle. If you still have your health (as far as you know), I guess you better try to enjoy the days you have left as they may come to an end quicker than you think.
Brilliant. Definitive. Reminds me of Dr. Geert Vanden Bossche's work, from a different angle. Thank you.