Weaponizing the Swarm by Weaponizing the Stupid

Why the suppression of "serial passage" as the technique used to craft SARS-CoV-2 depended on the cowardly compliance of morally and ethically bankrupt academic and journalistic worlds.

Robe and axe of Giovanni Bugatti, the official Papal Executioner who executed 514 people

Maybe the only thing less surprising than finding out that Tony Fauci, Jeremy Farrar, Kristian Andersen, Edward Holmes, and Ron Fouchier were extensively discussing serial passage as the most likely engineering technique used to create SARS-CoV-2 right at the outset of this pandemic, has been the fact that nobody in the corporate or pseudo-independent media has explored the implications of this technique being used in any way at all in the weeks since it was revealed.

For nearly three years now, I’ve been ignored and plagiarized by the corporate media, and almost entirely ignored by everyone else. 

Not one single person, calling themselves a “journalist” as they watch tens of millions of people die all around them, has expressed any interest at all in discussing anything that I’ve written. Actually that’s not true, interest was expressed by all of the following folks with very large voices, all of whom went on to subsequently ignore and often plagiarize my work: 

William J. Broad - New York Times

John Mecklin and Nicholson Wade - Bulletin of Atomic Scientists

Nicholson Baker - New York Magazine

Drew Hernandez - CNN

Josh Rogin - The Washington Post

Gary Ruskin - USRTK

David Malone - UK Channel 4

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Each and every one of you useless, dorky fucking assholes has either spoken with me directly, or cited my work inside your publications and then gone on to act like you don’t need to further cite them each and every time you pull from them. You do, that’s the basis of journalistic integrity, you lazy, idiotic, useless, cock-gargling, fucking fools. 

How much death does it take for any of you to discover ethics? 

Apparently there’s no answer to that, but here’s exactly what each and every one of you does need: To experience pain that you think is utterly limitless and will never stop, and to feel like your soul is drowning beneath a violence that will never end. 

Of course I mean that completely and entirely metaphorically, I’m not really saying that your role in perpetuating this unending wave of death and suffering means that you should literally be tortured to death by an angry mob with nothing on their minds but vengeance - only that your moral and intellectual bankruptcy is a direct result of the soft and easy lives each and every one of you has led. 

The Olympic Training Center at Colorado Springs

There’s a very profound difference between athletic and academic endeavors, probably no better captured than by how each field approaches dealing with the people who trained you. 

After discovering Canadian box lacrosse on a random cable-access channel and then finding out that field lacrosse was the only option in Maryland, and after realizing that as far as making a Varsity team went that I was way too fat and slow for soccer and too fat, slow, and short for basketball - I figured I saw a viable option within the little brother of warfare. I was generally more violent than those sports allowed, so lacrosse seemed to be a happy medium between running and beating people with a stick, but unluckily for my chonky fat ass - I wasn’t really fast enough to start on the high school lacrosse field either. 

If it hadn’t been for facing-off. 

Unlike hockey, where the puck drop happens in an instant, in lacrosse to start each quarter and after every goal, one player from each team would crouch down and fight for the ball like rabid seagulls after the last cracker at the ref’s whistle. Now here, here it didn’t matter how fast you’d been born or how long you’d been playing the sport - your stick started completely on the ground and literally backwards, this was completely and entirely about want to. 

But not just in the moment, it wasn’t enough to be determined and indomitable for those few moments fighting for the ball, the will to win begins with the will to prepare to win. So after touching the ball for maybe two entire minutes and failing to record a single assist or goal throughout my two years of middle school lacrosse at MVSA - incubator to all legendary lacrosse players - shortly before my freshman year of high school, I read online that wrestling gave face-off lacrosse guys a massive advantage, and I knew I’d found the next step. 

Luckily for me, the wrestling team was already training during my freshman JV football season, running literal circles around the bottom floor of our high school - very conveniently arranged into a circular shape that allowed athletes to have an indoor track with lots of very sharp turns. And so it wasn’t hard to find out about the team, and what I might be in for. 

Which is, as they say, a very long story. But after an uneventful freshman year wrestling, highlighted by our eventual state champ brandishing his mottled scrotum in my face and asking if I’d like some gum with hair in it, my sophomore year a new coaching staff showed up: Joe Hawkins and Coach Kim. Both in their mid-twenties and buddies since high school, alongside my wrestling partner, the late, great Greg Fisher, I got to experience something so much better than a storybook. 

Disney movies are fun for a few hours, but imagine sinking countless exhausted evenings at the gym alongside the best friend you’d ever have, and then suddenly those evenings merge into months and years, then in an instant you get to send him out onto the mat against a returning state qualifier two grades ahead of him, tasked by your coaches to get him ready for battle - pressing your foreheads together to scream into his eyes: YOU WIN, WE WIN!!

It was Fish’s first big Varsity match as a sophomore, and knowing I was about to wrestle some absolute turd who’d I was sure to pin and that our heavyweight would be a coin-flip, it was up to Fish to put the match away. And he did, pulling off the improbable victory as time ran out, and shocking everyone in the gym except me. 

Because by then I’d spent enough time watching him train in the gym, and then taking turns kicking his ass or getting my ass kicked in the wrestling room, depending on who was more tired at any particular moment. However when Fish won that huge match, vanquishing a foe two years his senior, neither of us had yet slayed each and every wrestler’s ultimate dragon: Scoring a takedown on one of our coaches. 

Unfortunately for Fred Kim, who’d had both his Achilles tendons severed when a forty-year old weight machine went tumbling over, snapping its cable which whipped across his heels and sliced away his ability to walk six months before the wrestling season, he went down first. But to be fair he hadn’t actually wrestled in college, just high school, and I was like 20 pounds bigger than him. 

So taking him down was a notch in my belt. But it wasn’t what I’d been training for. 

Now Farmer Joe, he’d wrestled at Old Dominion University, and been a state champ at the high school next door, and throughout my sophomore year when he first showed up trying to take him down felt like trying to fight my dad as a toddler. And so it would be many, many months of sweaty ass-whoopings in the wrestling room before I even sniffed grabbing his leg, nonetheless taking him all the way down to the ground. So it took awhile, right before the road to States would begin at the County Tournament a year later during my junior year, after well-over a year of trying - I finally got that takedown on Coach Joe Hawkins, a proud farmer who wasn’t too proud to make sure his athlete had a little confidence boost before the County tournament. 

Because although after I graduated Harvard and came back to work alongside him, he’d swear that takedown was legitimate - I never bought it. 

Since there’s an extraordinarily important and fundamental difference between training an athlete and training an academic, one that the fate of the entire world arguably hinges on. Training an athlete is about conflict, they’re going out to fight with an opponent, so as a coach it’s your job to prepare them for every possible occurrence that might get between them and victory. Coach Hawkins didn’t even have his right ACL anymore, but instead of getting surgery or worrying about his future mobility, he gritted his teeth and spent each and every single day on the mats with me and Fish, helping us chase our impossible dreams.

As a coach, as soon as you can, you teach your athletes each and every unlikely way things can go wrong, all of the possible adversity they might face as they stretch out to embrace their destiny - then as a coach, it’s your job to mimic that adversity in practice. Coaching an athlete in a combat sport is about kicking their ass as much as possible, preferably with your own hands, and hoping the day comes when they’ve got your number, and you have nothing left to give them. 

That’s your job as a coach, to lose, to be surpassed and eclipsed and bettered by someone, because you imparted everything you knew - dirty tricks and all - with the hope they’d use all you had to give on their way to victory against you. 

This dynamic stands in direct opposition to the academic world. 

Revenge of the Nerds

Unluckily for me, I got to Harvard in the wake of a massive grade inflation scandal, or at least massive as far as the Boston Globe went, since I had no idea at all when I got there. But it turns out that when you take a bunch of students with SAT scores around 1500/1600, it becomes really hard to give anyone anything less than A. Since everyone’s really smart, and they all do good work. 

But as our teaching fellows, Harvard’s bougie name for teaching assistants, explained to us my freshman year, in each reading section of about a dozen students, there’d be one A, one A-minus, and then everything else would be a B or lower. At that point I was on a full-ride from the NSA and just needed to keep a 3.0 for my scholarship, so I didn’t really care all that much at all beyond pride. 

However throughout my time at Harvard an important dynamic would play out: The students that got each A and A- weren’t doing anything different than their classmates beyond brown-nosing way more effectively. Unfortunately for my GPA it took until my sophomore spring until I realized that none of my professors were actually reading my papers or the essays I wrote during finals, instead they were being read by these teaching fellows, almost none of whom were really in no place at all to teach us anything since they were just a couple of years removed from where we were as students - not professors with decades of experience. 

But unlike the world of athletics, the academic world isn’t even vaguely a meritocracy. 

There’s very rarely any sort of demonstrated excellence, and in fact Scientific revolutions progress one death at a time largely because of this dynamic: Students are never, ever encouraged to challenge or defeat their teachers - Instead they’re trained to mindless stick their noses as far into their tangy bungholes as they possibly can. 

Because that’s what gets you that A, or the internship, or the overly flattering reference letter that gets you the job you don’t really have the merit for. Looking at you, every single paid “journalist” working for any corporate entity at all in America. 

And so the world has spent generations and generations in the ignorant mindless agreement of sissy scientists and academics who only ever learned how to snorkel way up inside their supervisors’ buttholes with their noses. But as one of the wisest men ever warned: The state which separates its scholars from its warriors, will have its thinking done by cowards and its fighting done by fools.

The History of the Peloponnesian War

And so as the Wuhan Strain of Coronavirus emerged in the winter of 2019, humanity was in for a very bad time. Because the decisions, those decisions would very much be made by cowards, and no fighting would happen at all because of those fools. America would decide to lay back, spread wide open like a Kardashian at Cannes, and let a reverting live-attenuated vaccine designed and distributed by China’s PLA infect nearly every citizen alive.

Because Tony Fauci and Jeremy Farrar, the latter being the WHO’s new Chief Scientist, consulted with Ron Fouchier and decided to perpetuate the cover-up that began with HIV, and occlude the origins of yet another live-attenuated vaccine (LAV) that reverted unexpectedly.  

After all, as the worst war criminals humanity will ever know are well-aware, live-attenuated vaccines can revert without too much trouble, as Ron Fouchier himself would explain in July 2019:

“That’s what happened in the 70s, people were trying to do live-attenuated vaccines and doing human challenge studies and that might be the way the H1 re-emerged in the 70s. Some people say it was a lab accident. I don’t believe that. I think it was actually human challenge studies and live-attenuated vaccines that reverted that are the likely candidates of the 1970 reemergence of H1. And we need to make sure that doesn’t happen again.”

However H1N1 is the highly-pathogenic state of human influenza, it is not an alien virus - it is completely and entirely adapted to our genome and has been with us for thousands of years. H1N1 doesn’t create a pandemic by simply by existing in a population, it is the strain that wins out and emerges once there’s enough crowding and transmission events to trick human influenza into thinking that its host population is about to die off completely, and so it goes into a highly-pathogenic state in an attempt to jump into a new host species, in its case from humans and into pigs.  

This is the exact same dynamic that plays out on poultry farms, as in those industrially crowded conditions the same dynamic emerges, and highly-pathogenic avian influenzas will begin to jump into the human workers who are in close and continuous contact with diseased flocks.

So it’s almost certainly the past reversions of H1N1 LAVs in 1977 and 2009 that seemed to eventually just melt away, which sociopaths like Richard Ebright and the rest of his sweaty socially-retarded buddies at JASON are using to assure everyone that SARS-CoV-2, another LAV that’s reverting, will just melt away in just a few more months - just like H1N1 seems to have done twice. And since SARS and MERS, artificial coronavirus chimeras that got out before deattenuation all the way down into LAVs, went away on their own - they think all signs point to that happening again.

Unfortunately, unlike their mythical buddy: Each and everyone one of these arrogant old hacks was drawn into the siren song of multi-billion dollar defense and pharmaceutical contracts long ago, and they’re going to remain pushing for a fascist and entirely ineffective vaccination program because they’re rotten, filthy, diseased whores, and that is exactly what they are being paid to do. 

Our novel coronavirus is not a naturally spreading and evolving virus, and it has not become endogenous to human populations after thousands of years of coevolution - it is reverting back towards a highly-pathogenic SARS-like chimera that our immune systems will be entirely helpless against, and is going through the same unexpected epistatic gatekeeping mutations that OPV does on its way back to full virulence as a VDPV, which vaccines are also entirely helpless against

In the case of SARS-CoV-2, this gatekeeping results in the sudden emergence of new strains that appear evolutionarily impossible - like Omicron.  And so long as transmission is ongoing, there is nothing that is going to stop this pandemic except more death, because transmission means more gatekeeping, and gatekeeping means continued steps closer to the original strongest version of this artificial highly-pathogenic virus. 

Being completely and entirely acclimated to the human genome is not at all the case with OPV, a LAV against the Polio virus that’s reverting into VDPV all across the third world and bringing back Polio’s terrible paralytic poliomyelitis. So OPV serves as a much more accurate analogy for SARS-CoV-2 than the H1N1 LAVs.

Our novel coronavirus is a LAV derived from the work being done at UNC, the only place on earth trying to make a LAV for SARS-like viruses, which are also obviously not going to be fully acclimated to the human genome like the human influenza virus, which seems to have been with us at least since the Trojan War thousands of years ago. 

Until SARS-CoV-2 is understood as a LAV that’s deattenuating towards a highly-pathogenic chimeric coronavirus that’s going through gatekeeping mutations and has no intention whatsoever of following the assumptions drawn from observing natural evolution or even the paths of the H1N1 LAVs which melted back into their original endogenous human hosts - humanity is going to continue to be standing on its head as it attempts to battle this pandemic, and misunderstanding the basic fundamental nature of what its up against. 

And the absolute worst part, is that all of this was totally avoidable and exposed just about in real time as it happened. Because what happened just two days after the absurdly flawed Proximal Origins paper came out back in February 2020? I responded with a takedown that entirely dissembled their paper, mostly using serial passage to poke holes in their absurd arguments. 

Then just a couple months later in April, with my dad’s help to polish the science, we submitted the paper that would pass peer-review and then get published in August 2020, outlining in very specific terms why serial passage could explain all of the anomalies. 

However maybe you’re new here and serial passage seems like somewhat of an esoteric term. So before looking at the newly FOIAed emails, here’s the explanation from our second peer-reviewed paper:

In the context of attenuating a potential vaccine candidate, serial passage refers to the process of allowing the natural and expected mutations that diminish a virus's virulence to occur—although in the unnatural setting of cell cultures, where recombination and other mutations are more likely—as it becomes acclimated to a new host species’ cells, which will have different receptor and backbone requirements than its original host. Once this initial adaptation occurs, initially yielding mutations that can either increase pathogenicity in the new host or be deleterious to it, additional “passages” in the form of the virus naturally passing among new hosts will then slowly lead to a natural attenuation of the virus relative to its old host. This is why passaging can be used to attenuate and weaken a virus in the effort to create vaccine candidates that will elicit immune responses, but less serious infections.

The other nominally related approach is serial passage through entire live hosts in a lab, which leads to a zoonosis that closely mirrors the natural process since the primary unnatural factor is logistical and not biochemical—simply host proximity and population density. This kind of forced zoonosis is not known to leave any signature other than an affinity for its original host; however, serial passage through cellular hosts that lack complete immune systems may leave its own signatures, because this type of passage simply cannot happen in nature. Natural zoonosis means passing the challenges naturally posed by the immune system of a new host species; however, cellular serial passage can bypass these processes and create unique phenomena, possibly like SARS-CoV-2's flat and non-sunken surface of its sialic acid-binding domain, which contrasts with the typical canyon observed in every single other ever observed coronavirus, as well as a lack of non-synonymous substitutions in its RBD, which would be expected to accumulate at some point during a virus's natural evoluation due to challenges from a novel host's complete immune system, but which would not be selected for and then observed to occur at the same frequency during cellular serial passage or during the few generations of whole host passaging required to establish airborne transmission.[2]

It gets a bit wonky at the end there, but the take-away is that even taking serial passage’s pseudo-natural nature into account - there’s a molecular signature of SARS-CoV-2 that indicates that its artificial. 

So with that in mind, maybe you can figure out why this entire section from the early drafts of Proximal Origins revealed in the newly FOIAed emails between Fauci and friends got removed?

Clearly, as Tony Fauci and friends were discussing at length in February 2020, the process of serial passage does in fact explain much of our friendly neighborhood novel coronavirus’s distinctive genome, as the godfather of the 2012 gain-of-function serial passage experiments with influenza and ferrets which also yielded an FCS, Ron Fouchier, who just mentioned that he thought the H1N1 outbreak was from a LAV, noted in those FOIAed emails:

If you’re familiar with our peer-reviewed papers, that will sound really familiar:

Several factors weigh into the probability that coronaviruses can gain furin cleavage sites following serial passage: The frequency of evolutionary motifs meant to deal with virus–host interactions that are often shared between viruses, the observations that when the infectious bronchitis coronavirus (IBV) coronavirus is serially passed through chickens it developed notable mutations along its spike‐protein genes,[ 21 ] and the fact that when a lineage A bovine coronavirus was subject to in vitro serial passage through cell lines, a 12‐nucleotide insert found within only a small minority of the pooled viruses spike‐protein region was strongly selected for and quickly emerged as the dominate strain.[ 22 ] These findings all point to the possibility that SARS‐CoV‐2 may have gained its furin cleavage site the same way influenza viruses do—through the in vivo serial passage between the live hosts that presents the immune challenges and intense selective pressure necessary for the recombination and mutations that lead to its emergence to occur.

SARS‐CoV‐2 has a remarkably strong affinity for spike‐protein binding to ACE2—some 10–20 times higher than SARS‐CoV's.[ 18 ] That affinity may have emerged after mutational events either in an intermediate natural host or after a zoonotic jump into humans that theoretically could have occurred earlier than the first documented infection, which would give it time to increase that significantly. So logically, it could also have emerged via selection after serial passage through laboratory cell cultures or laboratory animals as well.

Although it does not prove a laboratory origin, another gain‐of‐function experiment demonstrates one possible step along the way to engineering SARS‐CoV‐2: the synthetic reconstruction of the SARS coronavirus to impart this virus with a high affinity for ACE2. This involved isolating a progenitor coronavirus from civets and then serially passing it through mammalian ACE2 receptor‐expressing cells—serial passage through host cell lines instead of entire hosts, which imparted a strong affinity for ACE2,[ 35 ] and another novel strain of coronavirus that was also presumably airborne.

Seems a bit uncanny, right? Within those emails, Fouchier also asserts that betacoronaviruses can obtain an FCS from serial passage, something that elicits a lot of surprise from the WHO’s new Chief Scientist, Jeremy Farrar since it hadn’t been published yet:

“It’s what shifted me to thinking that the insert of the furin site is the result of cell culture passage (or less likely intense transmission in a non[-]bat host. Really need to see the data from Ron about generating the furin cleavage site on in vitro passage. Really!

Bottom line - I think if you put selection pressure on a Cov without a furin cleavage site in cell culture you could well generate a furin cleavage site after a number of passages (but let’s see the data Ron!)

So it must’ve been pretty baffling for these pretentious boffins as they watched my reply to their absurd Proximal Origins paper go up just 48 hours later, since I’d already ferreted out that serial passage explained all of this virus’s mysteries. And as the time’s passed I’ve really started to wonder why in the world I’m still alive, for at least the past year I’ve been explaining in very great detail how serial passage explained all of this, and why this technique was being suppressed since it unravels the entire conspiracy. 

Luckily for me, Nicholas Wade is a self-important twat who doesn’t know the first thing about opsec even though he’s been whitewashing this sort of virus-enhancing research for the government for literally 50 years. He slipped-up and dropped a very unusual nugget in his featured paper, while he was crediting someone else with being the first to outline why this virus looked engineered, and not mentioning our two peer-reviewed papers nor the fact me and my dad had been writing about this since January 2020 at all:

The first person to take a serious look at the origins of the SARS2 virus was Yuri Deigin, a biotech entrepreneur in Russia and Canada. In a long and brilliant essay, he dissected the molecular biology of the SARS2 virus and raised, without endorsing, the possibility that it had been manipulated. The essay, published on April 22, 2020, provided a roadmap for anyone seeking to understand the virus’s origins. Deigin packed so much information and analysis into his essay that some have doubted it could be the work of a single individual and suggested some intelligence agency must have authored it.

Bolded emphasis added since that essay, published months after I was writing about serial passage and being carried on Zero Hedge multiple times in the early months of 2020, only mentions serial passage once in passing. And it doesn’t echo with anything that’s mentioned in the newly FOIAed emails at all, he explores reverse genetics and waves his hands around as much as possible to distract from serial passage. Wade then goes on to mention a half-dozen other people as pioneering the exploration of a lab-engineered virus, none of them me or my father. 

Now why would that be? 

Because like Alina Chan before him, Nicholas Wade was happy to watch tens of millions of innocent strangers die terribly if it meant he got time in front of the cameras. And job number one was to make sure no discussion of serial passage ever surfaced at all, since it’s the hallmark of live-attenuated vaccines, and additionally points very directly back to the 2012 gain-0f-function experiments with ferrets and influenza that held the potential to “make the deadly 1918 pandemic look like a pesky cold.” 

Luckily for me, one day in March of 2020 I was feeling especially spicy for no particular reason at all, and decided to take a few swings and rattle a few cages. Although I’d been read-out of my TS/SCI clearance decades ago, I still remember all the fine print, and so when I passed all of our early work to my contact at the National Security Council, it was very intentionally through an intermediary. 

Because I didn’t really know what in the fuck was going on, but I’d already had some sockpuppets throw all sorts of weird, nonsensical, amateurish shit about the FBI at me - so I was going to be very careful not to accept nor solicit any sort of sensitive materials that could get me in trouble.

However that didn’t mean I couldn’t run a bluff:

Yeah. About all that. I’ve never had any contacts at the NSA looking into anything, and just about all of my contacts in the DC area treated me like I was dead when I was arrested, intelligence community or otherwise. 

No one other than my blood and a few former athletes looked me in the eye and welcomed me home after my release. 

So that entire thing was totally made up, but I’m pretty sure I made Andersen poop his lederhosen just a bit. And luckily for me and my dad, I’m sure it served as a red herring to get a whole bunch of idiots chasing someone leaking to me, since I’m just a criminal - HOW IN THE FUCK ELSE WOULD I BE CALLING ALL THEIR BULLSHIT BASICALLY IN REAL FUCKING TIME, HUH? 

Maybe because despite their prestigious offices, all of their fancy degrees and credentials and millions in stocks and royalties mean less than dust when it’s all said and done. All anyone had to do was read, simply read, and it’s all right there. From a word to a word, you’ll be led to a word. I haven’t formally studied biology since AP Bio over two decades ago, where I mostly remember trying to throw turtle testicles into my classmates’ mouths during dissections, but with my dad to guide me along I was able to pick all of this apart. 

Because I had no interest at all in mindlessly echoing Tony Fauci’s horrible murderous bullshit about a natural virus and a magical vaccine, nor did I care about the tide turning with the Proximal Origins paper, nor what anyone thought about an engineered virus being a “conspiracy theory.” Since I learned something very important from Farmer Joe on those wrestling mats - authority figures aren’t meant to be revered nor exalted, they are there for you to defeat them. 

Modern academia stultifies because all anyone gets rewarded for is upholding the status quo, and it’s populated by a bunch of conflict-avoidant dorks who can’t handle confrontation, and so they hide behind their obscure vocabularies and expensive degrees. And they continue to hide as tens of millions die all around them, and squeal and wave their hands when you point out their role in upholding the illusion allowing all of it to happen. 

Maybe there’s something to be said for the fact that if you locked me in a room with the roughly two-dozen pretentious fucks coordinating all of this while all of us were in our athletic primes, I’d systematically cut through all them inside about 15 minutes, killing or crippling all of them fairly easily. Maybe that sounds like a stretch, but in addition to writing most of this Wikipedia article, I spent two years training Kyle year-round and then spearheaded the effort to get him to the Olympic Training Center for his senior year of high school. And prior to that, after graduating college where I spent two years on the boxing club and before going back to coach wrestling at my old high school, I spent several months sparring with Darnell “Ding-a-Ling” Wilson, who would later go on to land what Teddy Atlas would call the most devastating knock-out in ESPN boxing history, who would’ve murdered me inside 45 seconds within a full fight, but who I learned enormously from within the limited sparring rules.

So please, please send someone after my person - hire some junkie to come after me and see what fucking happens.

You silly fucks think that hiding HIV fifty years ago means you can run the same playbook now and have it go off without a hitch, because all you’re used to is a bunch of pseudo-intellectual fuck-wits sucking you off and then snowballing your bullshit back onto the general public. 

Well, it worked until it didn’t. More FOIAs are coming, and hospitals in northern latitudes are already bursting at the seams. The truth is on it’s way out one way or another, and given what’s coming you’ve got to be doing the math and realizing that there are some little fish under you that you can sell on down the river to buy yourself some time. 

There’s already been an experimental finding dug-up that illustrates everything me and my dad got in the peer-reviewed literature regarding serial passage and FCS emergence was remarkably prescient:

Here we report that among spike protein sequences of genus Betacoronavirus and outside of the SARS-CoV-2 clade an analogous polyfunctional domain was found in only one other virus: an artificial MERS infectious clone, described already in 2017, which constitutes an adapted genotype from serial passage in genetically humanized mice. In contrast to this artificial MERS coronavirus, which has no natural primary host and which during passage acquired the full pat7 motif, S protein sequences from all other betacoronaviruses did not present a polyfunctional S1/S2 junction domain analogous to SARS-CoV-2. As the evolutionarily closest betacoronaviruses outside of the SARS-CoV-2 clade lack all its three functional features, this critical S1/S2 polyfunctional domain becomes an unlikely product of natural evolution alone.

Which is mirrored pretty uncannily by our August 2020 paper, that highlights the use of both humanized mice and ferrets for serial passage, and how convergent evolution meant one would likely emerge in betacoronaviruses with the proper evolutionary pressure. So going forward, it’s going to be pretty much impossible to publish either journalistically or academically without acknowledging that all of this was explained over two years ago in our peer-reviewed papers, and then further here. 

Impossible at least without really hilariously plagiarizing from this work even more, so feel free to have at it. But keep in mind, that America is already passed the event horizon of what used to be, and once the survivors understand what’s been so very preventably done to them, no one is going to be kept alive long enough to die of old age in a prison cell. 

Because this pandemic is very much just beginning, as I outlined in the whitepaper below, which was sent to every single government entity I’ve been in contact with since this started, and ignored by each and every one of them, from David Asher to the Chamber of Commerce and plenty in between. 

I am so very sorry to everyone that’s had to say goodbye, but I got buried a few years ago without even realizing it, by a community that decided that I no longer deserved any sort of a voice at all.

America likes to make a big deal about being the land of second-chances and redemption, however there’s another edge to that sword - The only reason redemption is such a big fucking deal in America, is because only in America is such a production made of judging and condemning those who dare stray out of line. There’s no redemption without judgement and condemnation, and it’s that Puritanical urge that arrived with us on these shores, to crush any sort of social dissent, that really drives the ethos of this broken, batshit nation.

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WEAPONIZING THE SWARM:

The Threat Posed by the Intentional Release of Targeted SARS-CoV-2 Variants

Although China’s insistence on anal swabs may seem puzzling to outsiders, because anal swabs are the only reliable way to monitor the full clearance of SARS-CoV-2 from hospital patients,[i] this allows the CCP to far more granularly monitor their infected communities. In addition to being the most accurate way to measure whether or not COVID-19 has cleared from a patient, anal swabs also show a much broader range of mutations and hence possible variants than nasal or pulmonary testing,[ii] each of which only reliably catch the variants acclimated to and living in those organ systems.

So by taking anal swabs, China has a much more granular picture of any infected communities, while nations predominately using nasal swabs will only be finding active nasal infections, since without an anal swab it is impossible to know if someone has truly cleared an infection. Additionally, nasal swabs give many false negatives for infections that anal swabs catch, since anal swabs catch both very weak infections, as well as display the full scope of variants that exist in any given host.

In January 2020, Dr. Charles Lieber was arrested for undisclosed financial arrangements with the CCP, and in addition to being the head of Harvard’s Chemistry Department, Dr. Lieber also pioneered the most sensitive viral variant technology systems every designed, much of which has been left unpublished for over a decade – however based on his charging documents, it can be surmised that nanotechnology was likely involved. It also follows that the initial ability to tell the difference between just broad classes of virus has likely become much more refined, and can likely detect individual SARS-CoV-2 variants, something that would explain whatever high-risk interaction led to his arrest, an odd moment at the start of the pandemic.

Another even more puzzling aspect of the genesis of this pandemic is the discrepancy between when the measurement of genetic change seems to indicate it started, roughly October 2019, and the fact that there was a happening around the WIV in December 2019. These two competing timelines are anchored by their assumed starting points, with the official reports from the data first released by China which is centered around the WIV and wetmarket representing December 2019 as the start of the outbreak.

However there is a second timeline, which is hinted at by the phylogenetic analysis which indicates that the outbreak actually started several months before, which meshes with the observation that the first patients actually appear to be at military hospitals farther north, and all anchoring on a subway line and the concomitant dense commercial setting.[iii] This timeline is also supported by evidence released by the State Department indicting a mysterious illness in and around Wuhan several months before December 2019.

One parsimonious explanation for this discrepancy would be if SARS-CoV-2 did in fact emerge in a diffuse manner throughout the commercial heart of Wuhan and not from any acute contact at all. Especially given how stealthily SARS-CoV-2 still spreads throughout new populations, largely asymptomatically and highly airborne, it seems reasonable to imagine that its initial emergence mirrored this diffuse and largely silent spread.

And so discovering a novel airborne RNA virus, the CCP may have applied some of the lessons learned attempting to vaccinate against another airborne coronavirus, Feline Infection Peritonitis Virus (FIPV), and attempted design a vaccine by removing select groups of genes. The authors of the paper explaining the approach observed that it “may allow the development of vaccines against infections with other pathogenic coronaviruses, including that causing severe acute respiratory syndrome in humans.”[iv]

However, the delicacy of vaccinating against airborne RNA viruses is also borne out by FIPV, since attempts to use vaccines only targeting the spike-protein can lead to early death syndrome in kittens. In fact, researchers have warned that:

“Since ADE in vitro has recently been described for human immunodeficiency virus type 1, current vaccination trials, particularly those aimed at raising an immune response against the [spike]-protein, should be reconsidered until more is known about the role of ADE in vivo.”[v]

So to avoid this phenomenon, instead of designing a vaccine that only targets the spike-protein, by deleting entire genes researchers hoped to created vaccines without any possible chance of increasing the virulence of an infection. However even here: “The accelerated development of disease in some of the animals suggested that the vaccination with this attenuated virus was detrimental rather than protective.” [vi]

Both of these well-established vaccination phenomena represent scenarios where the purported December 2019 outbreak localized around the WIV was not the start of China’s pandemic, but only the result of a vaccination trial with one of the two above strategies which backfired, resulting in the drastic measures taken by the CCP across Wuhan to enforce an extraordinary lockdown eventually encompassing tens of millions of its citizens.

If this is the case, then the emergence of the COVID-19 pandemic deep within Wuhan’s metropolis may not in fact have been natural, and may have been the result of the deattenuation of an experimental vaccine. In this scenario, the CCP knows exactly where SARS-CoV-2’s ongoing mutations are headed, which theoretically allows them to design attenuated variants that would mimic the unintentionally-lethal vaccine trials gone wrong.

And since America is only using nasal swabs to test and is not utilizing anal swabs, it would be impossible to detect the intentional release of these strains, designed to look natural but actually leading to increased-community wide lethality and death. Only anal swabs reveal the full extent of the variants within a population, and would allow for the detection of mathematically impossible mutations that these variants would have. And as Executive Orders warned: It is exceedingly easy to hide disguised aerosol dispersal devices in high-traffic commercial buildings, and now with WiFi ubiquitous the ability for targeted dispersal is even higher.   

Without anal swab data from some cross-section of the American population, it will be completely impossible to detect the release of such a weapon in an American urban center. And so long as policymakers are pushing for blanket vaccination programs which are not complimented by the data provided by anal swabs, our vaccination programs are at risk of eventually backfiring and leading to nearly instant death across wide swaths of the American population:

“The postmortem examination showed that the vFS-immunized kittens had suffered from a form of FIP much more severe than naturally occurring FIP, even though no gross changes were observed. The presence of multiple lesions in several organs is atypical of naturally occurring FIP and indicates a rapid dissemination. The relatively small size of the lesions is probably due to the short incubation period.” [vii]

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[i] Sun, Mei, et al. “Anal Swab as a Potentially Optimal Specimen for SARS-CoV-2 Detection to Evaluate Hospital Discharge of COVID-19 Patients.” Future Microbiology, Future Medicine Ltd, Aug. 2020, www.ncbi.nlm.nih.gov/pmc/articles/PMC7493721/.

[ii] Sun, Fengming, et al. “SARS-CoV-2 Quasispecies Provides Insight into Its Genetic Dynamics during Infection.” BioRxiv, Cold Spring Harbor Laboratory, 1 Jan. 2020, www.biorxiv.org/content/10.1101/2020.08.20.258376v1.full.

[iii] Quay, Dr. Steven. New Study By Dr. Steven Quay Concludes That SARS-CoV-2 Came from a Laboratory, 29 Jan. 2021, www.prnewswire.com/news-releases/new-study-by-dr-steven-quay-concludes-that-sars-cov-2-came-from-a-laboratory-301217952.html.

[iv] Haijema, Bert Jan, et al. “Live, Attenuated Coronavirus Vaccines through the Directed Deletion of Group-Specific Genes Provide Protection against Feline Infectious Peritonitis.” Journal of Virology, American Society for Microbiology Journals, 15 Apr. 2004, jvi.asm.org/content/78/8/3863.

[v] Vennema, H, et al. “Early Death after Feline Infectious Peritonitis Virus Challenge Due to Recombinant Vaccinia Virus Immunization.” Journal of Virology, American Society for Microbiology Journals, 1 Mar. 1990, jvi.asm.org/content/64/3/1407?ijkey=da9a99e3f31bfbf7a42a83a4667f4a339e333058&keytype2=tf_ipsecsha.

[vi] Haijema, Bert Jan, et al. “Live, Attenuated Coronavirus Vaccines through the Directed Deletion of Group-Specific Genes Provide Protection against Feline Infectious Peritonitis.” Journal of Virology, American Society for Microbiology Journals, 15 Apr. 2004, jvi.asm.org/content/78/8/3863.

[vii] Vennema, H, et al. “Early Death after Feline Infectious Peritonitis Virus Challenge Due to Recombinant Vaccinia Virus Immunization.” Journal of Virology, American Society for Microbiology Journals, 1 Mar. 1990, jvi.asm.org/content/64/3/1407?ijkey=da9a99e3f31bfbf7a42a83a4667f4a339e333058&keytype2=tf_ipsecsha.

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